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This is the first large study the authors could find that puts the question to a systematic test, using health records from Penn Medicine, an academic system covering more than 6.5 million patients around Philadelphia, Delaware, and southern New Jersey

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Under conditions of oxidant attack, GSH is rapidly converted to GSSG, making the elevation of the intracellular GSSG/GSH ratio a widely accepted marker for oxidative stress

This decrease was irrespective of cirrhosis etiology and thought to reflect diminished hepatic GSH synthesis

These improvements supported the healing of wounds infected with Staphylococcus aureus