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However, heterozygotes for CBS defects do not seem to have an increased risk for cardiovascular disease, because occurrence of these mutations is scarce in patients with vascular disease (Kluijtmans et al, 1996)

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aeruginosa , isolated from a mouse model of chronic infection, but originally from a CF patient, was performed to identify transcriptional modifications responsible for the phenotypic change from mucoid to small colonies vescicles (SCV)

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