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Furthermore, we note Y211C/H are variants associated with SPCDchanges that likely causes large structural perturbation of the carnitine-binding site beyond simply affecting solvent structure

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CPP-CAND disrupts FOXO4-p53 foci and induces apoptosis through caspase-dependent pathways FOXO4 is progressively recruited to euchromatin foci following senescence induction and is retained in the nucleus of senescent cells through its interaction with p53, contributing to the maintenance of the senescent state 34