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We found similar alterations in the probability of cellcell communication events between C35 GABA4 and C35 Glut10 sender neurons and hypothalamic receiver neurons after treatment with acyl-GLP-1 or the GIPR antagonist compared with vehicle DIO controls, which clearly diverge from that of acyl-GIP (Fig

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235:21-30 [PubMed: 24792990] 20

2022, Frontiers in Pharmacology )