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Erratum in: Nat Rev Gastroenterol Hepatol

Improving combination cancer therapy: the CombiPlex development platform

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While this effect typically diminishes as the body adjusts, it provides indirect evidence that the nitric oxide pathways are responding to the peptide

iii) Rab4A-driven mTOR activation promotes ANA production and GN, while the inactivation of Rab4A in T cells restrains CD98 expression, KYN accumulation, mTOR activation, mitochondrial metabolism and lineage skewing and blocks ANA and anti-phospholipid antibody (aPL) production and GN