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Only then can the cobalt atom be reduced from an oxidative state of +3 to the biologically active +1, forming methyl B12 in the cytosol, and adenosyl B12 in the mitochondria

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Patients with Leber's hereditary optic neuropathy should avoid cyanocobalamin (hydroxocobalamin is preferred in the UK)

In nature, the chemical form cyanocobalamin does not occur and must therefore be transformed in the body in order to take effect

The underlying logic is straightforward: if each component supports a different aspect of fat metabolism, combining all three could provide synergistic metabolic support

Periodic B12 and metabolic marker rechecking ensures continued safety