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Ycel B, Kelekci S, Demirel E

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Another study on mitochondria isolated from the forebrain of Fmr1 -KO mice confirmed the decreased respiratory function at complexes I and II, and the opening of the mPTP

ACOX and branch-chained acyl-CoA oxidase (another peroxisomal enzyme involved in FAO) mRNA levels were higher in patients with NAFLD compared to controls, indicating that peroxisomal FAO upregulation may be a compensatory response aiming to resolve the progressing steatosis in NAFLD [45, 107]

Age-matched db/+ mice served as a control group (Healthy control)

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