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[PubMed | ClinicalTrials.gov | DOI] Wadden TA, Bailey TS, Billings LK, et al

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J Biol Chem 279: 5534155347 Kogan I, Ramjeesingh M, Li C, Kidd JF, Wang YC, Leslie EM, Cole SPC, Bear CE (2003) CFTR directly mediates nucleotideregulated glutathione flux

Constitutive activation of NRF2, mainly via loss-of-function mutations in the CUL3-adapter KEAP1 or gain-of-function mutations in NFE2L2 , is thought to provide a fitness advantage to tumor cells in confronting metabolic/oxidative stress resulting from dysregulated growth (DeNicola et al, 2011