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Low apoptotic rates and overexpression of the anti-apoptotic gene, BCL2 , are considered as important pathogeneses of invasion and recurrence of keloids 5,31

As studies have stated 6 , alcohol consumption results in depletion of hepatic Glutathione, impairing antioxidant defence and increasing susceptibility to oxidative injury

10.1530/EC-19-0449 59 JinnoM.NagaiR.TakeuchiM.WatanabeA.TeruyaK.SugawaH.et al (2021)

Experimental models demonstrate that restoring glutathione normalizes mitochondrial redox status and improves endothelial and cardiovascular function, supporting a causal role for mitochondrial glutathione depletion in cardiovascular aging (17-18)