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Thus, it has been proposed that L-carnitine incr urea synth in the liver by facilitating fatty acid entry into mitochondria, leading to incr flux through the beta-oxidation pathway, an incr of intramitochondrial reducing equivalents, and enhancement of ATP production

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Its primary mechanism is VEGF upregulation, the growth of new blood vessels that supply oxygen and nutrients to injured tissue, alongside nitric oxide pathway modulation and fibroblast stimulation in tendon and ligament tissue