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Studies have demonstrated that in 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) and 6-Hydroxydopamine (6-OHDA)-induced PD animal models, the activation of microglia and the release of pro-inflammatory factors are key mechanisms underlying dopaminergic neuronal death

Specifically, we observed an upregulation of Glp1r (encoding glucagon-like peptide-1 receptor, GLP-1R), Htr4 (encoding 5-HT4 receptor), and Ntsr1 (encoding neurotensin receptor 1, NTR1)

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This concentration is specifically selected to meet USP monograph requirements for safety and efficacy in multi-dose vial applications

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Life at the edge: the nuclear envelope and human disease