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In laboratory animals, the forced overexpression of the TKT gene promotes the development of MAFLD as well as the advancement of MAFLD to the more severe state referred to as metabolic dysfunction-associated steatohepatitis (MASH)

In 2026, several Alzheimers and dementia treatments will enter their final crucible: Will they prove effective in slowing the disease, ease the symptoms, or simplify treatment

The imbalance of immune checkpoint signaling likely serves as a critical nexus in the cross-talk between ferroptosis and immune dysregulation, highlighting these molecules as potential therapeutic targets for combinatorial interventions targeting the ferroptosisimmunity axis in AD

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