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3.2.2 Inflammatory signaling pathways such as nuclear factor kappa B (NF-B) or p38 mitogen-activated protein kinase (MAPK) Targeting upstream master regulators of inflammation, such as the NF-B and p38 MAPK pathways, offers a strategic therapeutic approach by potentially suppressing a broad spectrum of downstream pro-inflammatory effectors implicated in tinnitus pathogenesis

Through incubation in fresh mouse serum, the results showed that the peptide modified with a C18 fatty diacid chain has a three-fold longer half-life in the serum compared to the peptide modified with a C10 fatty diacid chain

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Our previous data also suggests that maternal SE impairs brain mitochondrial levels of oxidative phosphorylation (OXPHOS) complexes in male offspring with reduced endogenous antioxidant capacity from birth (Chan et al., 2016b)

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Month 24: Final architecture demonstration and testing with documented details of the system or subsystem components being developed under the effort