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Subsequently, the activity of methylase increased, the expression of tumor suppressor Ras association domain family 1A (RASSF1A) was downregulated, and the ERK1/2 signaling pathway was activated, which regulated the activation of cardiac fibroblast proliferation and the transformation of endothelial cells into myofibroblasts [22]

Conditions less than ideal ( e.g

10.1016/j.tvjl.2012.05.027

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They use another protein, FOXO4, to keep p53 stuck in the nucleus so it cannot trigger cell death