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In the face of vitamin B 12 deficiency, conversion of methylmalonyl-CoA to succinyl-CoA cannot take place, which results in accumulation of methylmalonyl-CoA and aberrant fatty acid synthesis
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[23] Antivitamin B 12 compounds (often synthetic B 12 analogues) not only have no vitamin action, but also actively interfere with the activity of true vitamin B 12
Acute Fat Oxidation and Energy Expenditure Acute single administration of AOD9604 in wild-type mice produced a significant increase in fat oxidation and energy expenditure, with an associated reduction in glucose oxidation
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