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To evaluate differential expression (DE) attributable to hypoxia, HIF1 and HIF2 (EPAS1) knockdown, a trivariate model was applied using limma

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HIF-1 in a cellular microenvironment with low oxygen concentrations avoids being hydroxylated by prolyl 4-hydroxylases (PHDs) and subsequently degraded by the Von - Hippel -Lindau tumor suppressor protein (pVHL) [62]

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