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In conclusion, phytosterols and alkaloids can disrupt cancer-associated inflammatory and immune escape mechanisms by inhibiting tumor pro-inflammatory responses and enhancing antitumor immune effects, demonstrating potential clinical applications in improving the tumor microenvironment

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This in vitro model is nevertheless useful to distinguish between the effects of IGF-I on proliferation, differentiation and hypertrophy as IGF-I can be added or signalling can be modulated at different stages of the differentiation process