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While GHK has been shown to inhibit the inflammatory response and epithelial-mesenchymal transition (EMT) via the TGF-1/Smad 2/3 and IGF-1 pathways in bleomycin-induced fibrosis models [24], its broader anti-inflammatory effects in the context of pulmonary fibrosis pathogenesis may warrant further exploration

While redox homeostasis via enhanced glutathione production is commonly associated with the normal physiological role of NRF2, collectively our results suggest that NRF2-dependent cancer cell growth does not require this enhanced glutathione production

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Similar to AD and PD patients, approximately 90% of individuals with ALS are diagnosed with the sporadic form with no known gene mutations, and the remaining 10% of patients carry mutated genes

The Role of Antioxidants in the Chemistry of Oxidative Stress: A Review