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Of interest, treatment of aged rats with IGF-I was shown to upregulate eNOS and improve bioavailability of NO[295], and in vitro experiments in cultured endothelial cells demonstrated that IGF-I treatment reduce ROS production and upregulate eNOS[294]

However, little is known about the roles of these DA receptors in regulating -cell function

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In comparison to healthy subjects, there was a 3.5-fold increase in the burden of M