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Mice deficient in the S100A8/A9 heterodimer are protected from renal fibrosis following UUO treatment, indicating that S100A8/A9 mediates renal fibrosis, tubular apoptosis, and renal epithelial-mesenchymal transition post-UUO [263]

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In the realm of cancer imaging, GSH serves not only as a powerful endogenous stimulus for activation of imaging probes but also as a distinctive biomarker for malignant cells [19]

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