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these processes are mediated by PC1 and PC3.2830 OXM inhibits gastrointestinal secretion and motility (so probably promoting satiety) and stimulates pancreatic enzyme secretion and intestinal glucose uptake.31 Among others (cardiovascular and central effects), GLP-1 exerts a number of gastrointestinal actions: stimulation of glucose-induced insulin secretion by upregulating insulin gene expression and potentiating all steps of insulin biosynthesis

24,25 Treatment can also reduce lipid storage to protect against the development of fatty liver disease by inhibiting fatty acid synthesis and promoting fatty acid oxidation

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