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These changes can be categorized broadly into global/repetitive element methylation, gene-specific methylation, miRNA methylation, epigenetic clock genes alterations (see above), and methylation of immune- and retroviral-related elements 204

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GHK appears to directly target myofibroblasts, converting pathological persistent collagen deposition to physiological collagen contraction through ITG-1 activation and apoptotic elimination of excess myofibroblasts (Figure 5)

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