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Continuous disruption of the alveolar epithelial cell (AEC) layer promotes destruction of the basal membrane and the subsequent activation of an intraalveolar coagulation cascade, including imbalance of matrix metalloproteinases (MMPs) and tissue inhibitors of metalloproteinases (TIMPs), activation of myofibroblasts, ultimately results in the excessive deposition of extracellular matrix

Reported experience suggests that injury severity and how long the injury has lasted matter more than body weight

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Evidence shows that elevated intracellular GSH levels in some pathogens, such as P