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Defects in the OCTN2 gene cause primary carnitine deficiency, further emphasizing the strategic position of OCTN2 in maintaining carnitine homeostasis [165]

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Incretin therapies: highlighting common features and differences in the modes of action of glucagon-like peptide-1 receptor agonists and dipeptidyl peptidase-4 inhibitors

Rather than operating through a single pathway, the peptide influences multiple cellular systems that converge on tissue repair and regeneration

Until more robust evidence emerges, clinicians should continue following established anemia management guidelines and avoid assuming a direct therapeutic role for incretin-based therapies in iron disorders

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