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Experimental application must remain within controlled, in vitro environments
Our data indicate that the down-regulation of SLC25A39 results in ROS accumulation, while up-regulation of SLC25A39 decreases ROS production
Chen, R
The ischemia-reperfusion injury (I/R injury) of Sirt5 knockout mouse heart is restored to normal levels by dimethyl malonate (a succinate dehydrogenase (SDH) inhibitor) pretreatment, which implies that the change of SDH activity is the leading cause of the damage
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