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Civil Engineering, UCLA Last updated: July 2026 Human-researched and AI-assisted with full editorial review

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Furthermore, the identification of critical arginine demethylases that enable complete methylation and demethylation cycles will further elucidate virus-host interactions and expand the scope of regulatory mechanisms

Cell Death & Differentiation , 16(10): 1303-1314 James, S.J., Slikker, W

In the early stages of HSV-1 replication, only ATF6 was activated by proteolysis and its downstream targets, namely ER partners GRP78 and P94, were not upregulated, which enhanced the replication of HSV in cells and improved viral load (Burnett et al., 2012)

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