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Phosphorylation of HKIIs hydrophobic N-terminus enables outer mitochondrial membrane binding via voltage-dependent anion channel (VDAC) interaction, driving glycolytic flux while competitively inhibiting pro-apoptotic Bax-VDAC binding
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3e) was also reduced, as well as IL-1 protein (Fig
Age-Related changes in female murine reproductive mucosa with respect to T cell presence
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