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Cholinesterase inhibitors prescribed for dementia or mild Alzheimers disease keep acetylcholine in a neuronal synapse, as do medications treating myasthenia gravis and many eye conditions.42 Acetylcholine-influencing compounds are extremely common in nootropic supplements

For the myotendinous junction, as a combination point of anatomical and structural particularities (largely reviewed in [124,125]) such a new therapy possibility might be particularly important given the still unresolved problem of being the weakest element in the muscletendon unit, (and bonetendon junctions as extremely specialized tissues in general)

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doi: 10.1016/j.cell.2018.12.029 91 EgerodKLEngelstoftMSGrunddalKVNhrMKSecherASakataIet al

It is also described in mechanistic contexts involving nitric oxiderelated pathways, growth factor expression, and cellular processes tied to migration and adhesionconcepts often studied when researchers are investigating how tissues respond and reorganize after experimental injury or stress

Since its first description in the peer-reviewed literature by Neelakantan et al