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Song et al showed that decreasing periplasmic GSH by deleting the GSH exporter CydD triggers the CpxR/SoxS two-component systems, upregulating the AcrAB-TolC pump and lowering intracellular drug accumulation
Both pathways contribute to lipid peroxidation, with PUFA-related acyl-CoA esters serving as substrates
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different xenobiotics may induce distinct isoforms of the enzyme
