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Ryan, and P

Thus, these findings demonstrate the ability of TNBS to spontaneously complex free reduced GSH resulting in its almost instantaneous disappearance

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We first validated the buffering phenotype in the follow-up studies, in which SLC25A19 exhibited milder growth fitness defect compared to the control cells in the presence of complex III inhibitor antimycin, complex I inhibitor piericidin and complex V inhibitor oligomycin than in the regular culture condition (Supplementary Fig