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Interestingly, compared with classical SARS-CoV, ORF3a of SARS-CoV-2 can separate homotypic fusion and protein classification components, thereby inhibiting fusion of autophagosomes and lysosomes, which is a unique feature of SARS-CoV-2 inhibition of autophagy (212, 213)

Furthermore, HRMS-based non-targeted analyses using MS/MS structural elucidation allowed to identify other relatively minor metabolites related to APAP-Cys such as S-(5-acetylamino-2-hydroxyphenyl)mercaptopyruvic acid (SAMP), an APAP metabolite potentially formed by the transamination reaction of APAP-cysteine (see Fig.1), previously observed in mouse ( 29 )

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