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Intriguingly, the residues of c-MET II-SEMA that are involved in the binding to HGF I-K1 largely overlap with those at the c-MET I-SEMA/HGF I-K3 interface, suggesting that c-MET-SEMA cannot simultaneously bind to HGF-K1 and K3

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Lastly, we tested whether cells that were functionally deficient in mitochondrial ETC metabolism, owing to mutations in CI and CIV, were differentially sensitive to 5FU treatment