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BACH1 is a hemoglobin-binding transcription factor that plays a role in regulation of oxidative stress and iron metabolism

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To address the question about the relative contribution to anti-oxidant electron flux we made use of genetic or chemical inhibition of the glutathione and thioredoxin-dependent pathways and monitored the cytosolic NADP redox dynamics in response to exogenous oxidants in living cells and tissues

In vitro studies have also indicated that riboflavin itself has an antioxidant nature, independent of its action as the GR coenzyme ( Reference Toyosaki and Mineshita 47 )

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