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During ferroptosis, triggering stimuli can activate the process of ferritin degradation, termed ferritinophagy, or lead to the degradation of SLC40A1, thereby augmenting the levels of cytoplasmic Fe 2+ within unstable iron pools in cancer cells [104,105,106]

On contrary disulfides such as GSSG does not have any unique property that could be exploited for its quantification

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The study observed that GHK-Cu exposure possibly suppresses the activation of NF-B by modifying the phosphorylation of its inhibitor IB, thereby reducing the expression of inflammatory mediators

It is possible that the expected changed could have been identified should we have measured additional oxidative stress markers

J Appl Microbiol 132(1):390400