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Oxidative stress is implicated in the pathogenesis of SLE through multiple mechanisms, including mitochondrial dysfunction, activation of the type I IFN pathway, and the generation of oxidative DNA damage [130]

Alternatively, altered genetic regulation of Gpx4 expression caused by overexpression of mutated APP and PS1 in transgenic animals, may have disturbed the normal gene expression pattern, as it has been recently demonstrated to occur in the frontal cortex and hippocampus of transgenic mice carrying the E9 mutant hPS1 gene (Unger et al., 2005)

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The rate of increase in absorbance at 415 nm, which measures the reduction of 5-5-dithiobis (2-nitrobenzoic acid) by glutathione, reflects the total glutathione content (or oxidized glutathione when 2-VP is added)