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In vivo, GCLC overexpression significantly mitigated MPTP-induced motor deficits and dopaminergic neuron loss in mouse models

These discoveries will ultimately offer supporting evidence for clinical translation

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For instance, compound 6o was found to dose-dependently suppress lipopolysaccharide-induced TNF- generation in both in vivo and in vitro models, and the inhibition percentage of TNF- by compound 6o (100 mg/kg) in vivo reached 54.7%

Mitochondrial ROS regulate thermogenic energy expenditure and sulfenylation of UCP1