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Many of these neuroprotective effects of GLP-1 medicines in preclinical studies remain to be causally associated with the antiinflammatory actions of GLP-1 medicines (52)
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Here we determined the mechanisms and rate constants for the glutathione/glutaredoxin-dependent redox reactions of the widely used redox probe roGFP2 and addressed the question of how mono- and dithiol glutaredoxins catalyze the reduction of roGFP2(S 2 ) as a model non-glutathione protein disulfide substrate
The trials had high heterogenetiy ( I 2 = 71.2%), so the random-effects model was selected for meta-analysis
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