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Bertoni S, Albertini B, Facchini C, Prata C, Passerini N

doi: 10.1016/S1473-3099(11)70054-8 121 MunitaJ

doi: 10.2147/tcrm.s6178

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The peptide activates HGF/c-Met signaling cascades that upregulate synaptogenesis, dendritic spine growth, and NMDA receptor expression, and these changes continue autonomously once triggered

The positive outcomes of agents like canakinumab and colchicine suggest that effective strategies must precisely target the key nodessuch as the IL-1 pathway or the NLRP3 inflammasomethat directly perpetuate the vicious cycle between oxidative stress and inflammation detailed in Section 2.4.1