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Additionally, studies have indicated that inhibiting formation of new blood vessels and reducing permeability in subchondral bone can mitigate progression of OA (5,6)

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In both mouse models of autoimmune encephalomyelitis and human subjects, supplementation with zinc aspartate has been found to suppress the development of Th17 cells through the degradation of STAT-3, which may help alleviate the impact of Th17-driven autoimmune disorders [330, 331]

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