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Oxidative damage is widely recognized as a contributor to both aging and chronic disease, making this pathway particularly relevant to dermatology and longevity research

Increased programmed cell death of the syncytiotrophoblast disrupts the layer, resulting in homeostatic imbalance, which causes placental-derived material to be released into the maternal circulation, including tumor necrosis factor (TNF-alpha) and syncytiotrophoblast microparticles (SBTM) (51)

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