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Hodis E, Watson IR, Kryukov GV, Arold ST, Imielinski M, Theurillat JP, Nickerson E, Auclair D, Li L, Place C, Dicara D, Ramos AH, Lawrence MS, Cibulskis K, Sivachenko A, Voet D, Saksena G, Stransky N, Onofrio RC, Winckler W, Ardlie K, Wagle N, Wargo J, chong K, Morton DL, Stemk-Hale K, Chen G, Noble M, Meyrson M, Ladbury JE, Davies MA, Gershenwald JE, Wagner Sn, Hoon DSB, Shadendorf D, Lander ES, Gabriel SBl, Getz G, Garraway LA, Chin L

In summary, the pathomechanisms of acute COVID-19 infection in the CV system include direct viral toxicity, intracellular viral entry via ACE2 and glycocalyx modulation, alteration of ACE2 signaling cascade towards the activation of harmful Ang II/AT1R axis, activation of pro-inflammatory proteins and deregulation of the immune system, hyper-inflammatory milieu and cytokine storm formation, hyper-coagulopathy development, ED and damage (Figure 2), and cardiac fibrosis and cardiac remodeling (23, 114)

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