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when it is inhibited, neurons quickly restore glutamate neurotransmission by an ill-defined endogenous mechanism, suggesting that neurons might be making use of a storage buffer of glutamate (15)

Biological, physiological, pathophysiological, and pharmacological aspects of ghrelin

Preventing HGF nitration-dysfunction could also improve the therapeutic effects of investigational HGF drugs for various diseases including liver cirrhosis, chronic renal failure, lung fibrosis, myocardial infarction, arteriosclerosis obliterans, amyotrophic lateral sclerosis (ALS, Lou Gehrigs disease), and acute spinal cord injury 47,48,49,50,51,52,53,54,55

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Studies have shown significantly reduced GPX3 mRNA levels in chondrocytes from KBD patients, where elevated methylation of CpGs in the GPX3 gene suppresses its transcription, reducing antioxidant function and promoting chondrocyte apoptosis, thus accelerating KBD progression [205,206]

Domain I exhibits the structurally conserved thioredoxin-like fold motif (Figure 3A&C) [44, 68, 75]