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By promoting nitric oxide production, the peptide can improve endothelial function, reduce oxidative stress, and support angiogenesis

Importantly, these fat-reducing effects occurred without changes in lean tissue mass, suggesting selective targeting of adipose tissue

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B12 is a cofactor for the enzyme, methylmalonyl-CoA mutase, which transforms methylmalonyl-CoA into succinyl-CoA, which the body can directly use in the Krebs cycle to produce ATP

Monitoring for Response and Safety Monitor serum B12 levels periodically during therapy to establish adequacy of therapy

[PubMed] [Google Scholar] Gjurasin M., Miklic P., Zupancic B., Perovic D., Zarkovic K., Brcic L., et al