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Consistent with the metabolic improvements discussed previously, current evidence supports the premise that GLP-1RAs promote SHBG synthesis and reduce bioavailable free androgen activity primarily through weight loss and the alleviation of IR

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In chemotherapy-sensitive patients, Keap1 (Kelch-like ECH-associated protein 1) binds to NRF2 and mediates NRF2 ubiquitination via the Cul3-E3 ligase complex, promoting its degradation and maintaining NRF2 at low levels [32]

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