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Oxidative stress in hemodialysis: causative mechanisms, clinical implications, and possible therapeutic interventions

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In contrast, both hCiPSC-NSCs-treated rats (Fig

While this observational data has limitations and does not establish causation, the mechanistic basis for the difference is plausible: synthetic progestins like medroxyprogesterone acetate bind progesterone receptors with different affinity and also carry partial androgenic and glucocorticoid activity, whereas micronized progesterone behaves more selectively [5]

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LThyroxine induced hyperthyroidism effect on testicular endoplasmic reticulum stress and perkmediated Nrf2/HO1 signaling axis of rats