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Viral infections exacerbate FUS-ALS phenotypes in iPSC-derived spinal neurons in a virus species-specific manner

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During oxidative stress, the inability of Keap1 to bind NRF2 leads to NRF2 accumulation, which subsequently activates the expression of antioxidant genes

Despite this mechanistic insight, the clinical assessment of zinc status remains largely confined to systemic measurements, which poorly reflect intracellular distributions or organelle-specific dysfunction

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