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Sleep disorders, such as obstructive sleep apnea (OSA) and chronic insomnia have been implicated in an increased risk of ED [11,12,13,14,15,16], likely due to the cumulative effects of sleep fragmentation, intermittent hypoxia, and neuroendocrine disturbances [17]

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In this study, we aimed to analyze senescence during chondrocyte expansion and use senolytics to remove senescent chondrocytes for the enhancement of cartilage formation in ACI