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NNMT inhibition during this peak synthesis window allows more nicotinamide to enter the salvage pathway precisely when the cellular machinery is primed to convert it to NAD+

doi:10.1136/jcp.55.6.475

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Because NNMT activity intersects methyl donor utilization and nicotinamide handling, experimental inhibition can be used to probe pathway level relationships among methyl economy balance, NAD+ salvage flux, and downstream metabolic signaling in a range of preclinical systems

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