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[,] However, in inflammatory conditions, increased passage rates were observed in animal studies, suggesting that maternal comorbidities could potentially alter fetal exposure

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Extended prospective studies within non-cancer cohorts are essential to elucidate these risks

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Additionally, animal studies have found that exposure to glucagon-like peptide-1 receptor agonists in pregnancy resulted in adverse outcomes for offspring that included decreased fetal growth, skeletal and visceral anomalies, and embryonic death