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Melanotan II selectively activated hypothalamic oxytocin neurons and potentiated central oxytocin release in the nucleus accumbens, a brain region critical for social learning and reward

In our case, anti-VEGF therapy was started and tirzepatide was switched back to sitagliptin

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The central mechanism of action differentiates the compound from peripherally acting vasodilatory agents and has generated significant research interest in models of CNS-regulated physiological responses

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